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human prostate cancer pc3 cell lines  (Pasteur Institute)

 
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    Pasteur Institute human prostate cancer pc3 cell lines
    Human Prostate Cancer Pc3 Cell Lines, supplied by Pasteur Institute, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/pc3+prostate+cancer+cell+lines/pm42265189-37-6-15?v=Pasteur+Institute
    Average 86 stars, based on 1 article reviews
    human prostate cancer pc3 cell lines - by Bioz Stars, 2026-07
    86/100 stars

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    (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and <t>PC3</t> DM.
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    (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and <t>PC3</t> DM.
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    (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and <t>PC3</t> DM.
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    (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and <t>PC3</t> DM.
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    (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and PC3 DM.

    Journal: bioRxiv

    Article Title: Dynamic optimization of extrachromosomal DNA copy number drives tumour evolution

    doi: 10.64898/2026.03.20.713026

    Figure Lengend Snippet: (a) Schematics depicting hypothesized ecDNA copy number distributions of all cells, G1 cells, and parent cells giving rise to daughter cells (left). Representative images from combined IF for Aurora B Kinase and Cyclin A and DNA FISH for MYC in COLO 320DM. Cyclin A was used as a marker to identify G1 interphase cells, whose copy number should theoretically be 1n before entering S-phase and undergoing genome doubling. Aurora B Kinase was used as an identification marker for recently divided daughter cells. Copy number of parents of daughter cells was estimated by taking the average of the daughter cells’ MYC DNA FISH foci areas. Copy numbers of parent cells were compared against copy numbers of G1 interphase cells to avoid high copy number bias arising from cells past S-phase and with replicated DNA content. Scale bars, 10 µm (right). (b) Histograms depicting proportion of G1 interphase and parent cells in each copy number quintile in COLO 320DM and PC3 DM.

    Article Snippet: COLO 320DM and COLO 320HSR (colorectal cancer) and the parental PC3 (prostate cancer) cell lines were obtained from ATCC.

    Techniques: Marker